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The Year in Immunology: Basic and Clinical Research in Human Immunology, Volume 1285 / Edition 1
Barnes and Noble
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The Year in Immunology: Basic and Clinical Research in Human Immunology, Volume 1285 / Edition 1 in Chattanooga, TN
Current price: $144.50
Barnes and Noble
The Year in Immunology: Basic and Clinical Research in Human Immunology, Volume 1285 / Edition 1 in Chattanooga, TN
Current price: $144.50
Loading Inventory...
Size: OS
This volume of
The Year in Immunology
series features reviews of topics in human immunology, including B1 B cells, BCL-2 family members, Wiskott-Aldrich syndrome, reverse vaccinology, anti-b
2
-glycoprotein I, the role of autoimmunity In thromboangiitis obliterans (Buerger's Disease), long pentraxin PTX3 as a paradigm for humoral pattern recognition molecules, and control of inflammatory heart disease by CD4
+
T cells.
The Year in Immunology
series features reviews of topics in human immunology, including B1 B cells, BCL-2 family members, Wiskott-Aldrich syndrome, reverse vaccinology, anti-b
2
-glycoprotein I, the role of autoimmunity In thromboangiitis obliterans (Buerger's Disease), long pentraxin PTX3 as a paradigm for humoral pattern recognition molecules, and control of inflammatory heart disease by CD4
+
T cells.
This volume of
The Year in Immunology
series features reviews of topics in human immunology, including B1 B cells, BCL-2 family members, Wiskott-Aldrich syndrome, reverse vaccinology, anti-b
2
-glycoprotein I, the role of autoimmunity In thromboangiitis obliterans (Buerger's Disease), long pentraxin PTX3 as a paradigm for humoral pattern recognition molecules, and control of inflammatory heart disease by CD4
+
T cells.
The Year in Immunology
series features reviews of topics in human immunology, including B1 B cells, BCL-2 family members, Wiskott-Aldrich syndrome, reverse vaccinology, anti-b
2
-glycoprotein I, the role of autoimmunity In thromboangiitis obliterans (Buerger's Disease), long pentraxin PTX3 as a paradigm for humoral pattern recognition molecules, and control of inflammatory heart disease by CD4
+
T cells.
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